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Effect on breast tumors of DNA alternations in 3 genes described

block these receptors.

"Similarly, hypermethylation of E-cadherin was more frequent among progestin receptor (PR)-negative cases," said Tao. "Compared to tumors that were both ER- and PR-positive, tumors that were both ER- and PR-negative were more likely to be E-cadherin hypermethylated.

"However, hypermethylation of E-cadherin, p16 and RAR-â2 was not associated with other clinicopathological features of breast cancer, such as tumor size, histological grade or nuclear grade.

"Our data suggested that promoter hypermethylation is common in breast cancer," Tao added. "Because promoter hypermethylation is potentially reversible, identifying cancers with different hypermethylation may have important consequences for breast-cancer treatment."
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Source:University at Buffalo


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