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Story ideas from the Journal of Lipid Research
Date:8/27/2007

ulin production.

ER stress may also be reduced by targeting molecules involved in the UPR process. For example, a drug called Salubrinal was recently shown to inhibit one of the UPR-involved molecules and to protect cells from ER stress-induced cell death. Also, there is emerging evidence that anti-diabetic drugs may also work, at least in part, through this mechanism.

A deeper knowledge of how fat cells become dysfunctional will be critical in devising successful therapies in the future, the scientists conclude.

Article: Adipocyte stress: the endoplasmic reticulum and metabolic disease, by Margaret F. Gregor and Gokhan S. Hotamisligil

MEDIA CONTACT: Gokhan S. Hotamisligil, Harvard School of Public Health, Boston, Mass.; e-mail: ghotamis@hsph.harvard.edu


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Contact: Pat Pages
ppages@asbmb.org
301-634-7366
American Society for Biochemistry and Molecular Biology
Source:Eurekalert

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