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Researchers spot molecular control switch for preterm lung disorders
Date:3/20/2013

ouse BPD models. The drug, originally indicated to treat pain, protected the lungs from cell death, and was able to prevent destruction of and damage to the developing lung exposed to hyperoxia or excess interferon gamma in room air."

Bandari added that the findings suggest that Cox2 and or CHOP a molecule important in the stress pathway are potential new drug targets that can be inhibited to treat or prevent human BPD.

Bhandari said the next step is to conduct pre-clinical studies.


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Contact: Karen N. Peart
karen.peart@yale.edu
203-432-1326
Yale University
Source:Eurekalert  

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Researchers spot molecular control switch for preterm lung disorders
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