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Research offers insight into cellular biology of colorectal cancer
Date:8/21/2014

f moving parts, each doing something different and most still poorly understood," Neufeld said. "I think if the sole purpose of this protein was to target β-catenin for destruction, it wouldn't need to be this large. Our next job is to figure out exactly how goblet cell differentiation is controlled by one or more of APC's many components."

Beyond a slew of mechanistic details, the bigger picture that Neufeld and her group will keep exploring is that some colon cancers could arise from an inflammatory response to bacterial penetration of a thinner-than-normal mucus layer in the gut, resulting from defective APC. The possibilities of just what APC does and doesn't do, and how to compensate for any intestinal glitches related to loss of APC function, present a challenging mystery but also a plentiful harvest for scientists like Neufeld to reap going forward.


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Contact: Brendan M. Lynch
blynch@ku.edu
785-864-8855
University of Kansas
Source:Eurekalert  

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