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Rare gene glitch may hold clues for schizophrenia -- NIH-funded study
Date:2/24/2011

ential for treatment development, he added.

In addition to genetic screening of patients for such personalized medicine, Sebat sees opportunities in the new findings for neuroimaging studies. "We must determine how over-expression of VIPR2 impacts the growth, differentiation and function of neurons and how that influence behavior. Knowing where the VIPR2 gene is expressed in the human brain can point us to specific regions where VIP activity levels may differ between people who carry this mutation and those who don't."

Despite the challenges posed by the complexities of CNVs, Sebat is upbeat about prospects for ultimately making sense of the emerging evidence about schizophrenia genetics.

"We hypothesize that the many different genetic causes of schizophrenia may have something in common," he said. "There may be larger groups of patients who may not share the same mutation but, may share the same underlying neurobiological defect."


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Contact: Jules Asher
NIMHpress@nih.gov
301-443-4536
NIH/National Institute of Mental Health
Source:Eurekalert  

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