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Neurotoxins in shark fins: A human health concern
Date:2/23/2012

passing a wide range of neurological disorders. In 2009, Prof. Mash and her co-authors published a study in the journal Acta Neurological Scandinavica, demonstrating that patients dying with diagnoses of Alzheimer's Disease and ALS had unusually high levels of BMAA in their brains up to 256 ng/mg, whereas normal healthy aged people had no BMAA, or only trace quantities of the toxin present. "BMAA was first linked to neurodegenerative diseases in Guam, which resulted in the progressive loss of structure and function of neurons."

The shark study found a similar range and even higher BMAA in the fins tested. The new study found levels of between 144 and 1836 ng/mg of BMAA, which overlapped the levels we measured in the brains of Alzheimer's and ALS victims. Surprisingly, this level fits with the BMAA levels in fruit bats examined by Paul Cox, animals which concentrate BMAA from their diet of cycad seeds. He linked ingestion of fruit bats to the severe ALS/Parkinsonism dementia that afflicted many people in Guam.

"Not only does this work provide important information on one probable route of human exposure to BMAA, it may lead to a lowering of the demand for shark fin soup and consumption of shark products, which will aid ocean conservation efforts," added Hammerschlag.


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Contact: Barbra Gonzalez
barbgo@rsmas.miami.edu
305-421-4704
University of Miami Rosenstiel School of Marine & Atmospheric Science
Source:Eurekalert  

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