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Moleac Announces the Beneficial Effect of NeuroAiD (TM) (MLC 901) in Protecting the Brain Against Global Ischemia
Date:6/27/2011

SINGAPORE, June 27, 2011 /PRNewswire-Asia/ -- In June 2011, "Neuropharmacology" published a paper establishing NeuroAiD's reparative effects on global cerebral ischemia. Global cerebral ischemia is subsequent to cardiac arrest, reversible severe hypotension or other situations depriving the brain of oxygen and glucose. It results in residual neurologic deficits, ranging from mild cognitive impairment to severe neurological deficits, for which there is no treatment to date.

The research paper shows that NeuroAiD post global ischemia protects the neurons against ischemic injury in the CA1 region of the hippocampus which plays a critical role in the consolidation of information from short-term to long-term memory. In animal models, NeuroAiD inhibits neuronal cell death and protects neurons against DNA fragmentation by activating phosphorylation of the AKT protein and reducing oxidative stress. This corresponds to the better recovery of cognitive and motor deficits observed post stroke in clinical experiences.  

This study builds on previous research conducted in 2010 at the Institute of Molecular Pharmacology of the CNRS (French National Centre for Scientific Research) establishing NeuroAiD's neuroprotective and neuroreparative properties, and explaining the neurological recovery seen in post stroke patients.  The results established that NeuroAiD triggers the synthesis of the Brain Derived Neurotrophic Factor (BDNF), which is known to promote neuroplasticity. Indeed, NeuroAiD induces neurogenesis in rodent and human cells, and stimulates the development of new neuronal circuits of information by promoting cell proliferation and the development of dense axonal and dendritic networks. The recent data further confirms the neuroprotective effect of NeuroAiD and encourages prescribing it as soon as possible after a stroke so as to ensure a higher level of protection.

The lead researchers behind this new paper are
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SOURCE Moleac
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