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JCI table of contents: Feb. 21, 2011
Date:2/21/2011

rohn disease and that they do so via their adherence factor LPF. However, in an accompanying commentary, Warren Strober, at the National Institutes of Health, Bethesda, concludes that further work is needed to prove that AIEC are necessary for the initiation or persistence of Crohn disease.

TITLE: Crohn diseaseassociated adherent-invasive E. coli bacteria target mouse and human Peyer's patches via long polar fimbriae

AUTHOR CONTACT:
Arlette Darfeuille-Michaud
Clermont Universit, Clermont-Ferrand, Auvergne, France.
Phone: 33.4.73.17.79.97; Fax: 33.4.73.17.83.71; E-mail: arlette.darfeuille-michaud@u-clermont1.fr.

View this article at: http://www.jci.org/articles/view/44632?key=7de02b901f294f280590

ACCOMPANYING COMMENTARY
TITLE: Adherent-invasive E. coli in Crohn disease: bacterial "agent provocateur"

AUTHOR CONTACT:
Warren Strober
National Institute of Allergy and Infectious Diseases (NIAID), NIH, Bethesda, Maryland, USA.
Phone: 301.496.6810; Fax: 301.402.2240; E-mail: Wstrober@niaid.nih.gov.

View this article at: http://www.jci.org/articles/view/46333?key=6ad5cc80bfde0d210df8


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Contact: Ushma Neill
press_releases@the-jci.org
734-546-5241
Journal of Clinical Investigation
Source:Eurekalert

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