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JCI online early table of contents: Jan. 4, 2011

d Medical School, Boston, identified a new 18S E-series resolvin. Importantly, the new resolvin reduced levels of pro-inflammatory molecules and cells in a mouse model of inflammation caused by the bacterium E. coli. Further, as the new resolvin could be generated from aspirin, the authors suggest that this molecule could contribute to the beneficial effects of aspirin.

TITLE: Pro-resolving actions and stereoselective biosynthesis of 18S E-series resolvins in human leukocytes and murine inflammation

Charles N. Serhan
Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
Phone: 617.525.5001; Fax: 617.525.5017; E-mail:

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VACCINE DESIGN: How to keep fungi at bay

Infections with fungi that initially start at one site (often the lung) and then spread to other organs in the body are known as systemic fungal infections. As rates of such infections have increased dramatically in recent years, researchers are keen on developing vaccines against these invasive microbes. A team of researchers, led by Marcel Wthrich, at the University of Wisconsin, Madison, has now generated data in mice that provide insight into the type of immune response that such vaccines need to generate.

In North America, the three main fungi that cause systemic fungal infections are Coccidioides posadasii, Histoplasma capsulatum, and Blastomyces dermatitidis. In the study, immune cells known as Th17 cells were found to be sufficient for vaccine-induced protection against lethal lung infection with Blastomyces dermatitidis in mice. Further, vaccine-induced Th17 cells were necessary and sufficient to protect mice against all th

Contact: Karen Honey
Journal of Clinical Investigation

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