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Immune system release valve
Date:5/25/2011

self-destruct in a process called apoptosis. In the course of this process, the contents of the dying cells are prevented from spreading around and triggering inflammation. The present study, reported recently in Immunity, reveals an entirely new mechanism by which caspase-8 can control inflammation more directly. The research was performed in Wallach's lab by Dr. Akhil Rajput, Dr. Andrew Kovalenko, Dr. Konstantin Bogdanov, Seung-Hoon Yang, Dr. Tae-Bong Kang, Dr. Jin-Chul Kim and Dr. Jianfang Du.

The study results might be relevant for various types of inflammation not only that caused by viruses and can thus provide important insights into inflammatory disorders. Since such disorders may occur when the inflammatory response fails to be shut down properly, it's possible that caspase-8 malfunction and the resulting excessive activity of the RIP1 "signal amplifier" might be involved. And if this is indeed the case, a new treatment approach could aim at blocking RIP1, thereby fighting inflammation in a precise and selective manner.


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Contact: Yivsam Azgad
news@weizmann.ac.il
972-893-43856
Weizmann Institute of Science
Source:Eurekalert

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