Date:2/4/2011

often occurs in children and can be caused by genetic defects in factor H or in C3b, or else by the disruption of factor H activity by antibod-ies. In Finland, too, some of these patients have had to have complete liver-kidney transplants because of the severity of the disease. Consequently, the research's surprising and wide-reaching result will be important not only in terms of advancing basic immunological research but also in the diagnosis and treatment of very sick children.

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Contact: Dr. Sakari Jokiranta sakari.jokiranta@helsinki.fi 358-505-471-184 University of Helsinki Source:Eurekalert

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