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HIV study leads to insights into deadly infection
Date:7/8/2014

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"This suggests that you can't just prevent one part of SEVI from aggregating and being toxic to cells. You need to shut the whole thing down or stop it from forming in the first place," Dr Musgrave says.

Researchers also tested the fibrils against another major type of body tissue, epithelial cells, and found they were not toxic to these cells.

"Epithelial cells are a major barrier to HIV entry. There have been theories that the fibrils can damage the epithelial layer, making it much easier for the virus to enter the body and infect the immune cells, but our findings show that healthy epithelial cells are resistant," Dr Musgrave says.

"This is an important finding because it could mean that the toxicity from the fibrils is dependent on the type of tissue they come in contact with," Dr Musgrave says.

"We now have a better understanding of the role of these protein enhancers in HIV infection. However, it's clear that much more research is needed in this area," he says.


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Contact: Dr. Ian Musgrave
ian.musgrave@adelaide.edu.au
61-883-133-905
University of Adelaide
Source:Eurekalert

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