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Epilepsy drug shows potential for Alzheimer's treatment
Date:12/8/2008

ithin - and surmised that these changes might be involved in switching off NEP. The team found clear differences (acetylation) in key proteins within the chromatin when they compared normal nerve cells against those that failed to produce NEP.

"From there it was relatively simple to stimulate the expression of NEP with sodium valproate, which was seen to prevent the acetylation," says Professor Turner. "We were elated when we saw the results."


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Contact: Jo Kelly
jokelly@campuspr.co.uk
44-113-258-9880
University of Leeds
Source:Eurekalert

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