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Childhood virus infection linked to prolonged seizures with fever
Date:6/13/2012

who presented with FSE and received an assessment for herpesvirus infection within 72 hours of the episode. Viremia was detected using polymerase chain reaction (PCR) that identified the presence of HHV-6A, HHV-6B or HHV-7A DNA and RNA. In conjunction with PCR results, researchers used antibody titers to determine if the infection was a primary or reactivated herpesvirus.

Findings indicate that approximately one third of children with FSE had HHV-6 or HHV-7 viremia. HHV-6B viremia was detected in 32% of pediatric participants, with 38 and 16 children having primary and reactivated infection, respectively. Researchers found that 7% of children had HHV-7 viremia at baseline and 2 children had HHV-6/7 primary co-infection. There were no apparent differences in age, illness type, fever, seizure structures, or acute imagining abnormalities in children with or without one of the herpesviruses.

FEBSTAT researchers will continue to follow the 199 children involved in the study, expecting up to 40% of this pediatric population to develop TLE. The FEBSTAT study will determine whether there is an association between prolonged febrile seizures caused by HHV-6B viremia and the development of TLE. The current study adds to evidence from previous research by Donati et al. and by Fotheringham et al who detected HHV-6 in brain samples of patients with TLE.

"TLE could take 8 to 11 years to develop following an episode of FSE, so more time is needed before the role of HHV-6B is fully understood," concludes Dr. Epstein. "If the FEBSTAT study finds that FSE caused by HHV-6B leads to TLE, this insight would provide a basis for clinical trials of antiviral and anti-inflammatory therapies to prevent TLE."


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Contact: Dawn Peters
healthnews@wiley.com
781-388-8408
Wiley-Blackwell
Source:Eurekalert

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