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American Chemical Society's Weekly PressPac -- Aug. 15, 2007
Date:8/22/2007

s to complete properly, Cherkasov said. Finding an already existing, well-studied therapeutic agent that will kill an emerging bug might provide a rapid, first line of defense response option.

ARTICLE #2 EMBARGOED FOR: Sunday, Aug. 19, 2:45 p.m., Eastern Time

CONTACT:
Artem Cherkasov, Ph.D.
University of British Columbia
Vancouver, British Columbia V5Z 3J5
Canada
Phone: 604-875-4588
E-mail: artc@interchange.ubc.ca


ARTICLE #3 EMBARGOED FOR: Monday, Aug. 20, 10:15 a.m., Eastern Time

Common virus may contribute to obesity in some people

A common virus may be a contributing factor to the obesity epidemic sweeping through the United States and other countries, according to a new study by scientists in Louisiana. In laboratory experiments they showed that infection with human adenovirus-36 (Ad-36), long recognized as a cause of respiratory and eye infections in humans, transforms adult stem cells obtained from fat tissue into fat cells, whereas stem cells not exposed to the virus were unchanged, according to Magdalena Pasarica and colleagues.

The scientists also reported identification of a specific gene in the virus that appears to be involved in this obesity-promoting effect. The findings could lead to a vaccine or antiviral medication to help fight viral obesity in the future, the scientists said.

Were not saying that a virus is the only cause of obesity, but this study provides stronger evidence that some obesity cases may involve viral infections, Pasarica stated.

ARTICLE #3 EMBARGOED FOR: Monday, Aug. 20, 10:15 a.m., Eastern Time

CONTACT:
Magdalena Pasarica, M.D., Ph.D.
Louisiana State University
Baton Rouge, Louisiana 70803
Phone: 225-763-2778
E-mail: Magdalena.Pasarica@pbrc.edu


ARTICLE #4
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Contact: Michael Woods
m_woods@acs.org
202-872-4400
American Chemical Society
Source:Eurekalert

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